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CP-1 production \[[@pone.0173311.ref034]\] whereas, IL-1β is released from an inflammatory cell in the absence of the caspase-1 (i.e., inflammasome) \[[@pone.0173311.ref035]\]. This is consistent with our results, where a higher expression of *Tnf-α* was observed in TBV-infected sheep despite failing to show an increased level of NLRP3 inflammasome.

In this study, we observed a general increase in the expression of genes involved in iron metabolism and transport in the jejunal mucosa of sheep with experimental TBV infection. It is interesting to note that bovine TBV also induces hepcidin production *in vivo* \[[@pone.0173311.ref015]\] and our results are in agreement with this finding. Hepcidin blocks iron absorption and release of stored iron from macrophages, possibly protecting against infection by limiting iron availability for pathogens.